Search results for "Sotos syndrome"

showing 7 items of 7 documents

Incidentalome in Neurogenetics: Pathogenic Variant of NSD1 in a Patient With Spinocerebellar Ataxia (SCA)

2018

Background: Genetic studies of late-onset sporadic ataxias (>40 years of age) are not routinely indicated. For unresolved cases, next-generation sequencing (NGS) tools, such as whole-exome sequencing (WES), are available for a definitive diagnosis.Case presentation: Our patient is a woman with a usual facial phenotype and anthropometry, who developed ataxia at 45 years of age, with no relevant family history and an initial clinical approach that ruled out common aetiologies. WES was performed when the patient was 54 years old. The results identified the heterozygous pathogenic variant c.248delA (p.N83MfsX4) in the nuclear receptor-binding SET domain protein 1 (NSD1; MIM 606681) gene (rel…

0301 basic medicineAtaxialcsh:QH426-470Neurogeneticslate-onset sporadic ataxiasNSD103 medical and health sciencessymbols.namesakemedicineGeneticswhole-exome sequencingFamily historyGenetics (clinical)Exome sequencingGeneticsSanger sequencingSotos syndromebusiness.industrydiagnostics testmedicine.diseasePhenotypelcsh:Genetics030104 developmental biologyPerspectivegenetic incidentalomeSpinocerebellar ataxiasymbolsMolecular Medicinemedicine.symptombusinessFrontiers in Genetics
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Novel clinical findings in the first Egyptian case of Sotos syndrome caused by complete deletion of theNSD1gene

2017

0301 basic medicineGenetics03 medical and health sciences030104 developmental biologySotos syndromebusiness.industryGeneticsmedicinemedicine.diseasebusinessGeneGenetics (clinical)American Journal of Medical Genetics Part A
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Familial Sotos syndrome caused by a novel missense mutation, C2175S, in NSD1 and associated with normal intelligence, insulin dependent diabetes, bro…

2009

We report a familial Sotos syndrome in two children, boy and girl, aged 17 and 8 years, and in their 44 year old mother, who displayed normal intelligence at adult age, but suffered from insulin dependent diabetes mellitus, bronchial asthma, and severe lipedema. The underlying missense mutation, C2175S, occurred in a conserved segment of the NSD1 gene. Our findings confirm that familial cases of SS are more likely to carry missense mutations. This case report may prove useful to avoid underestimation of the recurrence rate of SS, and to demonstrate that the developmental delay may normalize, enabling an independent life and having an own family.

AdultMalemedicine.medical_specialtyPediatricsAdolescentLipid Metabolism DisordersMutation MissenseGermanyInternal medicineImmunopathologyGeneticsHumansMedicineMissense mutationGrowth DisordersGenetics (clinical)AsthmaAutoimmune diseaseType 1 diabetesbusiness.industrySotos syndromeRespiratory diseaseIntracellular Signaling Peptides and ProteinsLipoedemaNuclear ProteinsHistone-Lysine N-MethyltransferaseSyndromeGeneral Medicinemedicine.diseaseAsthmaDiabetes Mellitus Type 1EndocrinologyHistone MethyltransferasesFemalebusinessEuropean Journal of Medical Genetics
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To die or not to die ? un modèle de la signalisation dichotomique de TNF-R1

2004

1. Kurotaki N, Imaizumi K, Harada N, et al. Haplo insufficiency of NSD1 causes Sotos syndrome. Nature 2002 ; 30 : 305-6. 2. Douglas J, Hanks S, Temple K, et al. NSD1 mutations are the major cause of Sotos syndrome and occur in some cases of Weaver syndrome but are rare in other overgroth phenotypes. Am J Hum Genet 2003 ; 27 : 132-43. 3. Rio M, Clech L, Amiel J, et al. Spectrum of NSD1 mutations in Sotos and Weaver syndromes. J Med Genet 2003 ; 40 : 436-40. 4. Imaizumi K, Kimura J, Matsuo M, et al. Sotos syndrome associated with a de novo balanced reciprocal translocation t(5 ;8)(q35 ;q24.1). Am J Hum Genet 2002 ; 107 : 58-60. 5. Opitz JM, Weaver DW, Reynolds JF. The syndrome of Sotos and We…

Haplo insufficiencySotos syndromebusiness.industryHummedicineGeneral Medicinemedicine.diseasebusinessMolecular biologyGeneral Biochemistry Genetics and Molecular BiologyWeaver syndromemédecine/sciences
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Lymphoproliferative disorders in Sotos syndrome: Observation of two cases

1996

Sotos syndrome is included among the overgrowth disorders, most of which have an increased risk of neoplasms. Sotos syndrome does not appear to be related to a specific tumor type, but rather to the development of solid tumors of ectodermal or mesodermal origin in general. We report on two Sotos syndrome patients who developed a non-Hodgkin lymphoma and an acute lymphoblastic leukaemia, respectively. Our experience suggests that there may exist a high frequency of lymphoproliferative disorders in Sotos syndrome, and points out the importance of a long-term follow-up of Sotos syndrome patients, to detect a possible neoplastic evolution. ©1996 Wiley-Liss, Inc.

MalePathologymedicine.medical_specialtyLymphoproliferative disordersOvergrowth syndromeshemic and lymphatic diseasesmedicineHumansAbnormalities MultipleTumor typeSotos syndromeGrowth DisordersGenetics (clinical)Sotos syndromebusiness.industrySkullBrainSyndromemedicine.diseasePhenotypeLymphoproliferative DisordersLymphomaIncreased riskEl NiñoChild PreschoolLymphoblastic leukaemiabusinessAmerican Journal of Medical Genetics
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Deletion of NSD1 exon 14 in Sotos syndrome: first description.

2010

Settore MED/38 - Pediatria Generale E SpecialisticaSotos syndromeovergrowthmultiple ligation-dependent probe amplificationhuman genetics.
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Sotos syndrome and lymphoproliferative disorders: Reply to T. Cole and J. Allanson

1998

medicine.medical_specialtySotos syndromebusiness.industrymedicineLymphoproliferative disordersmedicine.diseasebusinessDermatologyGenetics (clinical)American Journal of Medical Genetics
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